The conducting state of TRPA1 modulates channel lateral mobility


Por: Sampieri A., Padilla-Flores T., Thawani A.R., Lam P.-Y., Fuchter M.J., Peterson R., Vaca L.

Publicada: 1 dic 2023 Ahead of Print: 1 sep 2023
Resumen:
We have studied Danio rerio (Zebrafish) TRPA1 channel using a method that combines single channel electrophysiological and optical recordings to evaluate lateral mobility and channel gating simultaneously in single channels. TRPA1 channel activation by two distinct chemical ligands: allyl isothiocyanate (AITC) and TRPswitch B, results in substantial reduction of channel lateral mobility at the plasma membrane. Incubation with the cholesterol sequestering agent methyl-beta-cyclodextrin (M beta CD), prevents the reduction on lateral mobility induced by the two chemical agonists. This results strongly suggest that the open conformation of TRPA1 modulates channel lateral mobility probably by facilitating the insertion of the channel into cholesterol-enriched domains at the plasma membrane.

Filiaciones:
Sampieri A.:
 Instituto de Fisiología Celular. Departamento de Biología Celular y del desarrollo. Universidad Nacional Autónoma de México. México, CDMX04510, Mexico

Padilla-Flores T.:
 Instituto de Fisiología Celular. Departamento de Biología Celular y del desarrollo. Universidad Nacional Autónoma de México. México, CDMX04510, Mexico

Thawani A.R.:
 Department of Chemistry, Molecular Sciences Research Hub, Imperial College London, White City Campus, London, W12 OBZ, United Kingdom

Lam P.-Y.:
 Department of Cell Biology, Neurobiology and Anatomy, Medical College of Wisconsin, 8701 West Watertown Plank Rd., Milwaukee, WI 53226, United States

 Neuroscience Research Center, Medical College of Wisconsin, 8701 West Watertown Plank Rd., Milwaukee, WI 53226, United States

Fuchter M.J.:
 Department of Chemistry, Molecular Sciences Research Hub, Imperial College London, White City Campus, London, W12 OBZ, United Kingdom

Peterson R.:
 College of Pharmacy, University of Utah, 30 South 2000 East, Salt Lake City, UT 84112, United States

Vaca L.:
 Instituto de Fisiología Celular. Departamento de Biología Celular y del desarrollo. Universidad Nacional Autónoma de México. México, CDMX04510, Mexico
ISSN: 01434160





Cell Calcium
Editorial
CHURCHILL LIVINGSTONE, JOURNAL PRODUCTION DEPT, ROBERT STEVENSON HOUSE, 1-3 BAXTERS PLACE, LEITH WALK, EDINBURGH EH1 3AF, MIDLOTHIAN, SCOT, Reino Unido
Tipo de documento: Article
Volumen: 116 Número:
Páginas:
WOS Id: 001097281300001
ID de PubMed: 37776645

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