Diabetes mellitus contribution to the remodeling of the tumor microenvironment in gastric cancer


Por: Rojas A., Lindner C., Schneider I., Gonzàlez I., Araya H., Morales E., Gómez M., Urdaneta N., Araya P., Morales M.A.

Publicada: 1 ene 2021
Resumen:
Compelling pieces of evidence derived from both clinical and experimental research has demonstrated the crucial contribution of diabetes mellitus (DM) as a risk factor associated with increased cancer incidence and mortality in many human neoplasms, including gastric cancer (GC). DM is considered a systemic inflammatory disease and therefore, this inflammatory status may have profound effects on the tumor microenvironment (TME), particularly by driving many molecular mechanisms to generate a more aggressive TME. DM is an active driver in the modification of the behavior of many cell components of the TME as well as altering the mechanical properties of the extracellular matrix (ECM), leading to an increased ECM stiffening. Additionally, DM can alter many cellular signaling mechanisms and thus favoring tumor growth, invasion, and metastatic potential, as well as key elements in regulating cellular functions and cross-talks, such as the microRNAs network, the production, and cargo of exosomes, the metabolism of cell stroma and resistance to hypoxia. In the present review, we intend to highlight the mechanistic contributions of DM to the remodeling of TME in GC. ©The Author(s) 2021. Published by Baishideng Publishing Group Inc. All rights reserved.

Filiaciones:
Rojas A.:
 Biomedical Research Lab., Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

Lindner C.:
 Biomedical Research Lab., Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

Schneider I.:
 Biomedical Research Lab., Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

Gonzàlez I.:
 Biomedical Research Lab., Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

Araya H.:
 Department of Clinical Sciences, Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

 Servicio de Oncología, Hospital Regional de Talca, Talca, 34600000, Chile

Morales E.:
 Biomedical Research Lab., Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

 Servicio de Anatomía Patologica, Hospital Regional de Talca, Talca, 34600000, Chile

Gómez M.:
 Department of Clinical Sciences, Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

 Servicio de Oncología, Hospital Regional de Talca, Talca, 34600000, Chile

Urdaneta N.:
 Department of Clinical Sciences, Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

 Servicio de Oncología, Hospital Regional de Talca, Talca, 34600000, Chile

Araya P.:
 Biomedical Research Lab., Medicine Faculty, Catholic University of Maule, Talca, 34600000, Chile

Morales M.A.:
 Department of Molecular and Clinical Pharmacology Program, Institute of Biomedical Sciences, University of Chile, Santiago, 8320000, Chile
ISSN: 19485204
Editorial
Baishideng Publishing Group, 7041 Koll Center Parkway, Suite 160, PLEASANTON, CA, UNITED STATES, Estados Unidos America
Tipo de documento: Article
Volumen: 13 Número: 12
Páginas: 1997-2012
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