Participation of Extracellular Vesicles from Zika-Virus-Infected Mosquito Cells in the Modification of Naive Cells' Behavior by Mediating Cell-to-Cell Transmission of Viral Elements


Por: Pablo Martinez-Rojas, Pedro, Quiroz-Garcia, Elizabeth, Monroy-Martinez, Veronica, Teresa Agredano-Moreno, Lourdes, Felipe Jimenez-Garcia, Luis, Ruiz-Ordaz, Blanca H.

Publicada: 1 ene 2020
Categoría: Biochemistry, genetics and molecular biology (miscellaneous)

Resumen:
To date, no safe vaccine or antivirals for Zika virus (ZIKV) infection have been found. The pathogenesis of severe Zika, where host and viral factors participate, remains unclear. For the control of Zika, it is important to understand how ZIKV interacts with different host cells. Knowledge of the targeted cellular pathways which allow ZIKV to productively replicate and/or establish prolonged viral persistence contributes to novel vaccines and therapies. Monocytes and endothelial vascular cells are the main ZIKV targets. During the infection process, cells are capable of releasing extracellular vesicles (EVs). EVs are mediators of intercellular communication. We found that mosquito EVs released from ZIKV-infected (C6/36) cells carry viral RNA and ZIKV-E protein and are able to infect and activate naive mosquito and mammalian cells. ZIKV C6/36 EVs promote the differentiation of naive monocytes and induce a pro-inflammatory state with tumor necrosis factor-alpha (TNF-alpha) mRNA expression. ZIKV C6/36 EVs participate in endothelial vascular cell damage by inducing coagulation (TF) and inflammation (PAR-1) receptors at the endothelial surface of the cell membranes and promote a pro-inflammatory state with increased endothelial permeability. These data suggest that ZIKV C6/36 EVs may contribute to the pathogenesis of ZIKV infection in human hosts.

Filiaciones:
Pablo Martinez-Rojas, Pedro:
 Univ Nacl Autonoma Mexico, Inst Invest Biomed, Dept Biol Mol & Biotecnol, Av Univ 3000,Ciudad Univ, Mexico City 04510, DF, Mexico

Quiroz-Garcia, Elizabeth:
 Univ Nacl Autonoma Mexico, Inst Invest Biomed, Dept Biol Mol & Biotecnol, Av Univ 3000,Ciudad Univ, Mexico City 04510, DF, Mexico

Monroy-Martinez, Veronica:
 Univ Nacl Autonoma Mexico, Inst Invest Biomed, Dept Biol Mol & Biotecnol, Av Univ 3000,Ciudad Univ, Mexico City 04510, DF, Mexico

Teresa Agredano-Moreno, Lourdes:
 Univ Nacl Autonoma Mexico, Fac Ciencias, Dept Biol Celular, Av Univ 3000,Ciudad Univ, Mexico City 04510, DF, Mexico

Felipe Jimenez-Garcia, Luis:
 Univ Nacl Autonoma Mexico, Fac Ciencias, Dept Biol Celular, Av Univ 3000,Ciudad Univ, Mexico City 04510, DF, Mexico

Ruiz-Ordaz, Blanca H.:
 Univ Nacl Autonoma Mexico, Inst Invest Biomed, Dept Biol Mol & Biotecnol, Av Univ 3000,Ciudad Univ, Mexico City 04510, DF, Mexico
ISSN: 20734409
Editorial
MDPI AG, ST ALBAN-ANLAGE 66, CH-4052 BASEL, SWITZERLAND, Suiza
Tipo de documento: Article
Volumen: 9 Número: 1
Páginas:
WOS Id: 000515398200123
ID de PubMed: 31947958

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