TSC2/PKD1 contiguous gene syndrome, with emphasis on a case with an atypical mild polycystic kidney phenotype and a novel genetic variant


Por: Reyna-Fabian, Miriam E., Alcantara-Ortigoza, Miguel A., Hernandez-Martinez, Nancy L., Berumen, Jaime, Jimenez-Garcia, Raquel, Gomez-Garza, Gilberto, Gonzalez-del Angel, Ariadna

Publicada: 1 ene 2020
Categoría: Nephrology

Resumen:
About 80% of patients with tuberous sclerosis complex (TSC) present renal involvement, usually as angiomyolipomas followed by cystic disease. An early diagnosis of polycystic kidney disease (PKD) in such patients is frequently related to the TSC2/PKD1 contiguous gene syndrome (PKDTS). Molecular confirmation of PKDTS is important for a prompt diagnosis, which can be complicated by the phenotypic heterogeneity of PKD and the absence of a clear phenotypegenotype correlation. Herein, we report three PKDTS pediatric patients. The case 3 did not present a classic PKDTS phenotype, having only one observable cyst on renal ultrasound at age 4 and multiple small cysts on magnetic resonance imaging at age 15. In this patient, chromosomal microarray analysis showed a gross deletion of 230.8 kb that involved TSC2, PKD1 and 13 other protein-coding genes, plus a heterozygous duplication of a previously undescribed copy number variant of 242.9 kb that involved six protein-coding genes, including SSTR5, in the 16p13.3 region. Given the observations that the case 3 presented the mildest renal phenotype, harbored three copies of SSTR5, and the reported inhibition of cystogenesis (specially in liver) observed with somatostatin analogs in some patients with autosomal dominant PKD, it can be hypothesized that other genetic factors as the gene dosage of SSTR5 may influence the PKD phenotype and the progression of the disease; however, future work is needed to examine this possibility. (C) 2019 Sociedad Espanola de Nefrologia. Published by Elsevier Espana, S.L.U.

Filiaciones:
Reyna-Fabian, Miriam E.:
 Secretaria Salud Mexico, Inst Nacl Pediat, Dept Genet Humana, Lab Biol Mol, Insurgentes Sur 3700-C, Mexico City 04530, DF, Mexico

Alcantara-Ortigoza, Miguel A.:
 Secretaria Salud Mexico, Inst Nacl Pediat, Dept Genet Humana, Lab Biol Mol, Insurgentes Sur 3700-C, Mexico City 04530, DF, Mexico

Hernandez-Martinez, Nancy L.:
 Secretaria Salud Mexico, Inst Nacl Pediat, Dept Genet Humana, Lab Biol Mol, Insurgentes Sur 3700-C, Mexico City 04530, DF, Mexico

Berumen, Jaime:
 Univ Nacl Autonoma Mexico, Fac Med, Dept Med Expt, Mexico City, DF, Mexico

 Hosp Gen Mexico City, Unidad Med Genom, Mexico City, DF, Mexico

Jimenez-Garcia, Raquel:
 Secretaria Salud Mexico, Inst Nacl Pediat, Serv Nefrol, Mexico City, DF, Mexico

Gomez-Garza, Gilberto:
 Secretaria Salud Mexico, Inst Nacl Ped, Resonancia Magnet, Mexico City, DF, Mexico

Gonzalez-del Angel, Ariadna:
 Secretaria Salud Mexico, Inst Nacl Pediat, Dept Genet Humana, Lab Biol Mol, Insurgentes Sur 3700-C, Mexico City 04530, DF, Mexico
ISSN: 02116995





Nefrologia
Editorial
SOC ESPANOLA NEFROLOGIA DR RAFAEL MATESANZ, HOSPITAL RAMON Y CAJAL CTR DE COLMENAR, KM 9,100, 28034 MADRID, SPAIN, España
Tipo de documento: Article
Volumen: 40 Número: 1
Páginas: 91-98
WOS Id: 000507855500011
ID de PubMed: 31176519

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