Mesenchymal Stromal Cells from the Epidermis and Dermis of Psoriasis Patients: Morphology, Immunophenotype, Differentiation Patterns, and Regulation of T Cell Proliferation


Por: Castro-Manrreza, M. E., Bonifaz, L., Castro-Escamilla, O., Monroy-Garcia, A., Cortes-Morales, A., Hernandez-Estevez, E., Hernandez-Cristino, J., Mayani, H., Montesinos, J. J.

Publicada: 1 dic 2019
Resumen:
Psoriasis is a skin disease characterized by hyperproliferation of keratinocytes and chronic inflammation. Mesenchymal stem/stromal cells (MSCs) exhibit an immunoregulatory function that can be altered in the skin of these patients. However, to date, the presence and functional capacity of MSCs in the dermis and epidermis of patients with psoriasis have not been fully established. In the present study, we evaluated the presence of MSCs in the skin of patients by obtaining adherent cells from the dermis and epidermis of lesional and nonlesional areas and characterizing them in a comparative manner with corresponding cells obtained from the dermis (HD-MSCs) and epidermis (HE-MSCs) of healthy donors. We determined whether the adherent cells had immunophenotypic profiles and differentiation potentials that were characteristic of MSCs. In addition, we analyzed their immunosuppression function by evaluating their capacity to decrease T cell proliferation. Our results indicate the presence of MSCs in the dermis and epidermis of healthy donors and patients with psoriasis; adherent cells from all skin sources exhibited MSC characteristics, such as expression of CD73, CD90, and CD105 markers and a lack of hematopoietic and endothelial marker expression. However, the cell populations obtained showed differences in differentiation potential toward adipogenic, osteogenic, and chondrogenic lineages. In addition, we observed a low MSC obtention frequency in nonlesional epidermal samples (NLE-MSCs), which also showed alterations in morphology and proliferation rate. Interestingly, MSCs from both the nonlesional dermis (NLD-MSCs) and lesional dermis (LD-MSCs) showed higher HLA class I antigen (HLA-I) expression than HD-MSCs. Moreover, NLD-MSCs showed a low T cell proliferation suppression capacity. In summary, this study demonstrates the presence of MSCs in the epidermis and dermis of patients with psoriasis and suggests that such cells may favor the inflammatory process and thus psoriatic lesion development through high HLA-I expression and low immunosuppression capacity.

Filiaciones:
Castro-Manrreza, M. E.:
 Univ Nacl Autonoma Mexico, Lab Inmunol & Celulas Troncales, FES Zaragoza, Unidad Multidisciplinaria Invest Expt Zaragoza, Mexico City, DF, Mexico

Bonifaz, L.:
 Inst Mexicano Seguro Social, Ctr Med Nacl Siglo 21, Hosp Especialidades, Unidad Invest Med Inmunoquim, Mexico City, DF, Mexico

Castro-Escamilla, O.:
 Inst Mexicano Seguro Social, Ctr Med Nacl Siglo 21, Hosp Especialidades, Unidad Invest Med Inmunoquim, Mexico City, DF, Mexico

Monroy-Garcia, A.:
 Inst Mexicano Seguro Social, Ctr Med Nacl Siglo 21, Lab Inmunol & Canc, Unidad Invest Med Enfermedades Oncol, Mexico City, DF, Mexico

Cortes-Morales, A.:
 Inst Mexicano Seguro Social, Ctr Med Nacl Siglo 21, Lab Celulas Troncales Mesenquimales, Unidad Invest Med Enfermedades Oncol, Mexico City, DF, Mexico

Hernandez-Estevez, E.:
 Inst Mexicano Seguro Social, Ctr Med Nacl Siglo 21, Lab Celulas Troncales Mesenquimales, Unidad Invest Med Enfermedades Oncol, Mexico City, DF, Mexico

Hernandez-Cristino, J.:
 Inst Mexicano Seguro Social, Ctr Med Nacl Siglo 21, Lab Celulas Troncales Mesenquimales, Unidad Invest Med Enfermedades Oncol, Mexico City, DF, Mexico

Mayani, H.:
 Inst Mexicano Seguro Social, Ctr Med Nacl Siglo 21, Lab Celulas Troncales Hematopoyet, Unidad Invest Med Enfermedades Oncol, Mexico City, DF, Mexico

Montesinos, J. J.:
 Inst Mexicano Seguro Social, Ctr Med Nacl Siglo 21, Lab Celulas Troncales Mesenquimales, Unidad Invest Med Enfermedades Oncol, Mexico City, DF, Mexico
ISSN: 1687966X





Stem Cells International
Editorial
Hindawi Publishing Corporation, 410 PARK AVENUE, 15TH FLOOR, #287 PMB, NEW YORK, NY 10022 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 2019 Número:
Páginas:
WOS Id: 000503497000003
ID de PubMed: 31885608

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