The role of glycolysis-derived hexose phosphates in the induction of the Crabtree effect


Por: Lemus M.R., Roussarie E., Hammad N., Mougeolle A., Ransac S., Issa R., Mazat J.-P., Uribe-Carvajal S., Rigoulet M., Devin A.

Publicada: 17 ago 2018
Resumen:
Evidence for the Crabtree effect was first reported by H. Crabtree in 1929 and is defined as the glucose-induced decrease of cellular respiratory flux. This effect was observed in tumor cells and was not detected in most non-tumor cells. A number of hypotheses on the mechanism underlying the Crabtree effect have been formulated. However, to this day, no consensual mechanism for this effect has been described. In a previous study on isolated mitochondria, we have proposed that fructose-1,6-bisphosphate (F1,6bP), which inhibits the respiratory chain, induces the Crabtree effect. Using whole cells from the yeast Saccharomyces cerevisiae as a model, we show here not only that F1,6bP plays a key role in the process but that glucose-6-phosphate (G6P), a hexose that has an effect opposite to that of F1,6bP on the regulation of the respiratory flux, does as well. Thus, these findings reveal that the Crabtree effect strongly depends on the ratio between these two glycolysis-derived hexose phosphates. Last, in silico modeling of the Crabtree effect illustrated the requirement of an inhibition of the respiratory flux by a coordinated variation of glucose-6-phosphate and fructose-1,6-bisphosphate to fit the respiratory rate decrease observed upon glucose addition to cells. In summary, we conclude that two glycolysis-derived hexose phosphates, G6P and F1,6bP, play a key role in the induction of the Crabtree effect.

Filiaciones:
Université Bordeaux, IBGC, UMR 5095, Bordeaux Cedex, France
Institut de Biochimie et Génétique Cellulaires, CNRS UMR 5095, 1, rue Camille Saint Saëns, Bordeaux Cedex, France
Instituto de Fisiología Celular, Universidad Nacional Autónoma de México, Mexico City, Mexico
Université Bordeaux, IBGC, UMR 5095, Bordeaux Cedex, 33077, France
Institut de Biochimie et Génétique Cellulaires, CNRS UMR 5095, 1, rue Camille Saint Saëns, Bordeaux Cedex, 33077, France
Instituto de Fisiología Celular, Universidad Nacional Autónoma de México, Mexico City, 04510, Mexico
ISSN: 00219258
Editorial
AMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INC, 9650 ROCKVILLE PIKE, BETHESDA, MD 20814-3996 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 293 Número: 33
Páginas: 12843-12854
WOS Id: 000442203500017
ID de PubMed: 29907566

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