Insular cortex lesions impair the acquisition of conditioned immunosuppression


Por: Ramírez-Amaya V., Alvarez-Borda B., Ormsby C.E., Martínez R.D., Pérez-Montfort R., Bermúdez-Rattoni F.

Publicada: 1 ene 1996
Resumen:
Conditioned immunosuppression can be readily obtained in animals by associating a taste with an immunosuppressive drug. On subsequent exposure to the conditioned taste, the animals show an attenuated immune response and also exhibit a conditioned taste aversion. It has been established that insular cortex lesions disrupt the acquisition of conditioned taste aversion. The effect of NMDA-induced lesions in either the insular cortex or the parietal cortex of male Wistar rats was evaluated in the acquisition of conditioned immunosuppression in two experiments. Unlesioned control rats showed the conditioned immunosuppressive response after reexposure to the taste, as indicated by lower hemagglutinating titers to sheep red blood cells in the first experiment and by a decreased IgM production to ovalbumin, measured by ELISA, in the second experiment. Insular cortex-lesioned rats did not show the conditioned immunosuppression in either experiment, while parietal cortex lesions and the sham-lesioned animals presented a clear decrease of hemagglutinating titer and a low IgM production. The insular cortex lesions did not affect the normal immune response, showing normal hemagglutinating titers and IgM production when compared to nonconditioned controls. The immunosuppressive action of cyclophosphamide also remained unaltered. In conclusion, these results show that the insular cortex is essential for the acquisition of conditioned immunosuppression.

Filiaciones:
Ramírez-Amaya V.:
 Departamento de Neurociencias, Facultad de Psicología, Univ. Nac. Autonoma de Mexico, 04510 México, D.F., Mexico

 Depto. de Psicofisiología, Facultad de Psicología, Univ. Nac. Autonoma de Mexico, 04510 México, D.F., Mexico

Alvarez-Borda B.:
 Departamento de Neurociencias, Facultad de Psicología, Univ. Nac. Autonoma de Mexico, 04510 México, D.F., Mexico

Ormsby C.E.:
 Departamento de Neurociencias, Facultad de Psicología, Univ. Nac. Autonoma de Mexico, 04510 México, D.F., Mexico

Martínez R.D.:
 Departmento de Med. Exp. de la F., Facultad de Psicología, Univ. Nac. Autonoma de Mexico, 04510 México, D.F., Mexico

Pérez-Montfort R.:
 Depto. de Microbiologia del I., Facultad de Psicología, Univ. Nac. Autonoma de Mexico, 04510 México, D.F., Mexico

Bermúdez-Rattoni F.:
 Departamento de Neurociencias, Facultad de Psicología, Univ. Nac. Autonoma de Mexico, 04510 México, D.F., Mexico

 Departamento de Neurociencias, Inst. de Fisiología Celular, Univ. Nac. Autonoma de México, P.O. Box 70-253, 04510 México, D.F., Mexico
ISSN: 08891591
Editorial
Academic Press Inc., 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 10 Número: 2
Páginas: 103-114
WOS Id: A1996VF83600003
ID de PubMed: 8811934

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