A study of the process of apoptosis in animals infected with the contagious ecthyma virus


Por: Garrido-Farina, GI, Cornejo-Cortes, MA, Martinez-Rodriguez, A, Reyes-Esparza, J, Alba-Hurtado, F, Tortora-Perez, J

Publicada: 25 may 2008
Resumen:
Contagious ecthyma virus (CEV) is a disease caused by a parapoxvirus, also is a potent genetic carrier with the capacity for regulating apoptosis in the cells of infected skin, a mechanism that serves for evading the immune response of the host. It has been suggested that the virus may remain in the skin and be able to cause repeated infections in the same flock. The effect of infection as well as the presence of contagious ecthyma virus was evaluated in terms of lesions and apoptosis in the skin of animals, infected both naturally and experimentally. Samples used were obtained from a naturally infected sheep, 5 goats inoculated with CEV and a negative control. Samples obtained were longitudinally sectioned and processed using photon and electron microscopy, and embedded in paraffin and araldite. Samples embedded in paraffin were sectioned in 5 mu m of thickness and dyed with orange eosin-hematoxilin G and Gomori's trichrom stain, apoptosis was demonstrated by the TUNEL assay, the vira

Filiaciones:
Garrido-Farina, GI:
 FES Cuautitlan Univ Nacl Autonoma Mexico, Dept Biol Sci, Morphol Sci Sect, Support Lab Histol & Biol, Mexico City, DF, Mexico

Martinez-Rodriguez, A:
 FES Cuautitlan Univ Nacl Autonoma Mexico, Dept Biol Sci, Multidisciplinary Unit Res Anim Hlth, Mexico City, DF, Mexico

Reyes-Esparza, J:
 Autonomous Univ, Fac Pharm, Cytometry Unit, Mexico City, DF, Mexico

Alba-Hurtado, F:
 FES Cuautitlan Univ Nacl Autonoma Mexico, Dept Biol Sci, Multidisciplinary Unit Res Anim Hlth, Mexico City, DF, Mexico

Tortora-Perez, J:
 FES Cuautitlan Univ Nacl Autonoma Mexico, Dept Biol Sci, Multidisciplinary Unit Res Anim Hlth, Mexico City, DF, Mexico
ISSN: 03781135





VETERINARY MICROBIOLOGY
Editorial
Elsevier, PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS, Países Bajos
Tipo de documento: Article
Volumen: 129 Número: 1-2
Páginas: 28-39
WOS Id: 000255990700003
ID de PubMed: 18191506

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