Mutants of the Zebrafish K+ Channel Hcn2b Exhibit Epileptic-like Behaviors


Por: Rodriguez-Ortiz, Roberto, Matinez-Torres, Ataulfo

Publicada: 1 nov 2021
Resumen:
Epilepsy is a chronic neurological disorder that affects 50 million people worldwide. The most common form of epilepsy is idiopathic, where most of the genetic defects of this type of epilepsy occur in ion channels. Hyperpolarization-activated cyclic nucleotide-gated (HCN) channels are activated by membrane hyperpolarization, and are mainly expressed in the heart and central and peripheral nervous systems. In humans, four HCN genes have been described, and emergent clinical data shows that dysfunctional HCN channels are involved in epilepsy. Danio rerio has become a versatile organism to model a wide variety of diseases. In this work, we used CRISPR/Cas9 to generate hcn2b mutants in zebrafish, and characterized them molecularly and behaviorally. We obtained an hcn2b mutant allele with an 89 bp deletion that produced a premature stop codon. The mutant exhibited a high mortality rate in its life span, probably due to its sudden death. We did not detect heart malformations or important heart rate alterations. Absence seizures and moderate seizures were observed in response to light. These seizures rarely caused instant death. The results show that mutations in the Hcn2b channel are involved in epilepsy and provide evidence of the advantages of zebrafish to further our understanding of the pathogenesis of epilepsy.

Filiaciones:
Rodriguez-Ortiz, Roberto:
 Cátedras CONACyT—Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Campus UNAM-Juriquilla, Universidad Nacional Autónoma de México, Querétaro, CP 76230, Mexico

 Univ Nacl Autonoma Mexico, Catedras CONACyT, Dept Neurobiol Celular & Mol, Inst Neurobiol, Campus UNAM Juriquilla, Queretaro 76230, Mexico

Matinez-Torres, Ataulfo:
 Departamento de Neurobiología Celular y Molecular, Instituto de Neurobiología, Campus UNAM-Juriquilla, Universidad Nacional Autónoma de México, Querétaro, CP 76230, Mexico

 Univ Nacl Autonoma Mexico, Dept Neurobiol Celular & Mol, Inst Neurobiol, Campus UNAM Juriquilla, Queretaro 76230, Mexico
ISSN: 16616596





INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Editorial
MDPI AG, POSTFACH, CH-4005 BASEL, SWITZERLAND, Suiza
Tipo de documento: Article
Volumen: 22 Número: 21
Páginas:
WOS Id: 000719376500001
ID de PubMed: 34768904
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