Association of TLR4 gene polymorphisms with sepsis after a burn injury: findings of the functional role of rs2737190 SNP


Por: Colin-Castro, Claudia A., Franco-Cendejas, Rafael, Rocha-Gonzalez, I, Hector, Cruz-Arenas, Esteban, Leyva-Garcia, Norberto, Sanchez-Sanchez, Roberto, Leyva-Gomez, Gerardo, Gomez, Rocio, Munoz, Balam, Cortes, Hernan, Magana, Jonathan J.

Publicada: 1 may 2021 Ahead of Print: 1 feb 2021
Resumen:
Sepsis is a life-threatening organ dysfunction condition caused by a dysregulated response to an infection that is common among patients with moderate to severe burn injury. Previously, genomic variants in Toll-like receptor 4 (TLR4), a key innate immunity receptor, have been associated with sepsis and infection susceptibility. In this study, the association of six TLR4 SNPs with sepsis after burn injury was tested in the Mexican mestizo population. We found that the rs2737190 polymorphism is associated with sepsis after burn trauma. Interestingly, the G allele and GG genotype were associated with a lower risk of developing sepsis. Since the rs2737190 SNP is in the promoter region of the TLR4 gene, we analyzed the possibility that this polymorphism regulates the TLR4 pathway. We cultured peripheral blood mononuclear cells from different genotype carriers and found, after stimulation with LPS, that carriers of the GG genotype showed a higher expression of TLR4, IL6, and TNF alpha than AA genotype carriers. The results suggest that the GG genotype produces an increase in the TLR4 expression, and therefore an improvement in the immune response. We conclude that the rs2737190 polymorphism may become a useful marker for genetic studies of sepsis in patients after a burn injury.

Filiaciones:
Colin-Castro, Claudia A.:
 Rocha-Gonzalez, Hector, I, Inst Politecn Nacl, Escuela Super Med, Secc Estudios Posgrad & Invest, Mexico City, DF, Mexico

 Inst Nacl Rehabil Luis Guillermo Ibarra Ibarra IN, Natl Ctr Res & Care Burns CENIAQ, Dept Genet, Lab Genom Med, Mexico City, DF, Mexico

Franco-Cendejas, Rafael:
 INR LGII, CENIAQ, Dept Infectol, Mexico City, DF, Mexico

Cruz-Arenas, Esteban:
 INR LGII, Epidemiol Surveillance Unit, Mexico City, DF, Mexico

Leyva-Garcia, Norberto:
 Inst Nacl Rehabil Luis Guillermo Ibarra Ibarra IN, Natl Ctr Res & Care Burns CENIAQ, Dept Genet, Lab Genom Med, Mexico City, DF, Mexico

Sanchez-Sanchez, Roberto:
 INR LGII, Dept Biotechnol, CENIAQ, Mexico City, DF, Mexico

Leyva-Gomez, Gerardo:
 Univ Nacl Autonoma Mexico, Chem Fac, Dept Pharm, Mexico City, DF, Mexico

Gomez, Rocio:
 CINVESTAV IPN, Dept Toxicol, Mexico City, DF, Mexico

Munoz, Balam:
 Sch Engn & Sci, Dept Bioengn, Tecnol Monterrey Campus Ciudad Mexico ITESM CCM, Mexico City, DF, Mexico

Cortes, Hernan:
 Inst Nacl Rehabil Luis Guillermo Ibarra Ibarra IN, Natl Ctr Res & Care Burns CENIAQ, Dept Genet, Lab Genom Med, Mexico City, DF, Mexico

Magana, Jonathan J.:
 Inst Nacl Rehabil Luis Guillermo Ibarra Ibarra IN, Natl Ctr Res & Care Burns CENIAQ, Dept Genet, Lab Genom Med, Mexico City, DF, Mexico

 Sch Engn & Sci, Dept Bioengn, Tecnol Monterrey Campus Ciudad Mexico ITESM CCM, Mexico City, DF, Mexico
ISSN: 14664879





GENES AND IMMUNITY
Editorial
NATURE PUBLISHING GROUP, MACMILLAN BUILDING, 4 CRINAN ST, LONDON N1 9XW, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 22 Número: 1
Páginas: 24-34
WOS Id: 000614015300001
ID de PubMed: 33531683