Abnormalities in subsets of B and T cells in Mexican patients with inborn errors of propionate metabolism: observations from a single-center case series


Por: Medina-Torrez, E. A., Vela-Amieva, M., Galindo-Campos, L., Ibarra-Gonzalez, I., Espinosa-Padilla, S., Guillen-Lopez, S., Lopez-Mejia, L., Fernandez-Lainez, C.

Publicada: 1 ene 2021
Resumen:
Background: Propionate inborn errors of metabolism (PIEM), including propionic (PA) and methylmalonic (MMA) acidemias, are inherited metabolic diseases characterized by toxic accumulation of propionic, 3-hydroxypropionic, methytcitric, and methylmalonic organic acids in biological fluids, causing recurrent acute metabolic acidosis events and encephalopathy, which can lead to fatal outcomes if managed inadequately. PIEM patients can develop hematological abnormalities and immunodeficiency, either as part of the initial clinical presentation or as chronic complications. The origin and characteristics of these abnormalities have been studied poorly. Thus, the aim of the present work was to evaluate and describe lymphoid, myeloid, and erythroid cell population profiles in a group of clinically stable PIEM patients. Methods: This was a retrospective study of 11 nonrelated Mexican PIEM patients. Clinical, biochemical, nutritional, hematological, and lymphocyte subsets were analyzed. Results: Despite being considered clinically stable, 91% of patients had hematological or immunological abnormalities. The absolute lymphocyte subset counts were tow in all patients but one, with CD4+ T-cell lymphopenia, being the most common one. Furthermore, of the 11 studied subjects, nine presented with a low CD4/CD8 ratio. Among the observed hematological alterations, bicytopenia was the most common (82%) one, followed by anemia (27%). Conclusion: Our results contribute to the landscape of immunological abnormalities observed previously in PIEM patients; these abnormalities can become a life-threatening chronic complications because of the increased risk of opportunistic diseases. These findings allow us to propose the inclusion of monitoring immune biomarkers, such as subsets of lymphocytes in the follow up of PIEM patients. (C) 2021 Codon Publications. Published by Codon Publications.

Filiaciones:
Medina-Torrez, E. A.:
 Inst Nacl Pediat, Secretaria Salud, Unidad Invest Inmunodeficiencias, Mexico City, DF, Mexico

Vela-Amieva, M.:
 Secretaria Salud Mexico, Lab Errores Innatos Metab & Tamiz, Inst Nacl Pediat, Mexico City, DF, Mexico

Galindo-Campos, L.:
 Secretaria Salud Mexico, Lab Errores Innatos Metab & Tamiz, Inst Nacl Pediat, Mexico City, DF, Mexico

 Univ Nacl Autonoma Mexico, UNAM, Fac Quim, Mexico City, DF, Mexico

Ibarra-Gonzalez, I.:
 Univ Nacl Autonoma Mexico, Inst Invest Biomed, Inst Nacl Pediat, Unidad Genet Nutr, Mexico City, DF, Mexico

Espinosa-Padilla, S.:
 Inst Nacl Pediat, Secretaria Salud, Unidad Invest Inmunodeficiencias, Mexico City, DF, Mexico

Guillen-Lopez, S.:
 Secretaria Salud Mexico, Lab Errores Innatos Metab & Tamiz, Inst Nacl Pediat, Mexico City, DF, Mexico

Lopez-Mejia, L.:
 Secretaria Salud Mexico, Lab Errores Innatos Metab & Tamiz, Inst Nacl Pediat, Mexico City, DF, Mexico

Fernandez-Lainez, C.:
 (Corresponding Author), Inst Nacl Pediat, Lab Errores Innatos Metab & Tamiz, Torre Invest,Av IMAN 1,Piso 9, Alcaldla Coyoacan 04530, Cdmx, Mexico

 Secretaria Salud Mexico, Lab Errores Innatos Metab & Tamiz, Inst Nacl Pediat, Mexico City, DF, Mexico
ISSN: 03010546
Editorial
ELSEVIER DOYMA SL, TRAVESERA DE GARCIA, 17-21, BARCELONA, 08021, SPAIN, España
Tipo de documento: Article
Volumen: 49 Número: 1
Páginas: 101-106
WOS Id: 000619192200014
ID de PubMed: 33528936