Treatment with Hyaluronic Acid and Collagen-Polyvinylpyrrolidone Improves Extracellular Matrix Assembly for Scarring after Tracheal Resection


Por: Olmos-Zuniga, J. Raul, Baltazares-Lipp, Matilde, Hernandez-Jimenez, Claudia, Jasso-Victoria, Rogelio, Gaxiola-Gaxiola, Miguel, Silva-Martinez, Mariana, Iniguez-Garcia, Marco Antonio, Gonzalez-Gonzalez, A. Ivan, Vazquez-Minero, Juan Carlos, Luna-Flores, Antonia, Solis-Alanis, Norma, Baltazares-Lipp, Mario Enrique

Publicada: 27 ago 2020
Resumen:
Treatment of tracheal stenosis is occasionally performed in combination with wound healing modulators to manipulate new extracellular matrix (ECM) formation and prevent fibrosis. Hyaluronic acid (HA) and collagen-polyvinylpyrrolidone (collagen-PVP) decrease fibrosis in experimental tracheal healing. However, they have not been used clinically as their effect on ECM components, which modify tracheal scarring, has not been described.Objective. To evaluate the effect of the application of HA, collagen-PVP, a mixture of HA and collagen-PVP (HA+collagen-PVP), and mitomycin C on the expression of decorin, matrix metalloproteinase 1 (MMP1), and MMP9, as well as the type of collagen and deposits formed in the scar after resection and end-to-end anastomosis (REEA) of the cervical trachea using an experimental model.Materials and Methods. Thirty dogs underwent REEA of the cervical trachea and were treated with different wound healing modulators: group I (n=6), control; group II (n=6), HA; group III (n=6), collagen-PVP; group IV (n=6), HA+collagen-PVP; and group V (n=6), mitomycin C. The dogs were evaluated clinically and endoscopically for 4 weeks. Subsequently, macroscopic and microscopic changes, expression of ECM proteins, and collagen deposition in tracheal scars were analysed.Results. Groups II, III, and IV showed reduced endoscopic, macroscopic, and microscopic inflammation, improved neovascularization, high decorin expression (p<0.01, analysis of variance (ANOVA)), and moderate expression of MMP1 (p<0.003, ANOVA) and type I and III collagen (p<0.05, Kruskal-Wallis). Groups IV and V developed fewer collagen deposits (p<0.001, ANOVA).Conclusion. Treatment with HA and collagen-PVP improved post-REEA healing by increasing neovascularization, stimulating the expression of decorin, and regulating the expression of MMP1, as well as type I and III collagen and their deposition.

Filiaciones:
Olmos-Zuniga, J. Raul:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Dept Surg Res, Lung Transplantat Res Unit, Mexico City, DF, Mexico

Baltazares-Lipp, Matilde:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Dept Surg Res, Mexico City, DF, Mexico

Hernandez-Jimenez, Claudia:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Dept Surg Res, Mexico City, DF, Mexico

Jasso-Victoria, Rogelio:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Dept Surg Res, Mexico City, DF, Mexico

Gaxiola-Gaxiola, Miguel:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Lab Morphol, Mexico City, DF, Mexico

Silva-Martinez, Mariana:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Dept Surg Res, Lung Transplantat Res Unit, Mexico City, DF, Mexico

Iniguez-Garcia, Marco Antonio:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Subdirect Surg, Mexico City, DF, Mexico

Gonzalez-Gonzalez, A. Ivan:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Dept Otolaryngol Head & Neck Surg, Mexico City, DF, Mexico

Vazquez-Minero, Juan Carlos:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Subdirect Surg, Mexico City, DF, Mexico

Luna-Flores, Antonia:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, Dept Surg Res, Lung Transplantat Res Unit, Mexico City, DF, Mexico

Solis-Alanis, Norma:
 Univ Nacl Autonoma Mexico, Fac Med Vet & Zootecnia, Dept Surg, Mexico City, DF, Mexico

Baltazares-Lipp, Mario Enrique:
 Inst Nacl Enfermedades Respiratorias Ismael Cosio, External Consultat Serv, Mexico City, DF, Mexico
ISSN: 23146133
Editorial
HINDAWI PUBLISHING CORPORATION, 410 PARK AVENUE, 15TH FLOOR, #287 PMB, NEW YORK, NY 10022 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 2020 Número:
Páginas:
WOS Id: 000570885900003
ID de PubMed: 32908887