DNA methylation in the pathogenesis of polycystic ovary syndrome


Por: Ricardo Vazquez-Martinez, Edgar, Ines Gomez-Viais, Yadira, Garcia-Gomez, Elizabeth, Reyes-Mayoral, Christian, Reyes-Munoz, Enrique, Camacho-Arroyo, Ignacio, Cerbon, Marco

Publicada: 1 jul 2019
Resumen:
Polycystic ovary syndrome (PCOS) is the leading endocrine and metabolic disorder in premenopausal women characterized by hyperandrogenism and abnormal development of ovarian follicles. To date, the PCOS etiology remains unclear and has been related to insulin resistance, obesity, type 2 diabetes mellitus, cardiovascular disease and infertility, among other morbidities. Substantial evidence illustrates the impact of genetic, intrauterine and environmental factors on the PCOS etiology. Lately, epigenetic factors have garnered considerable attention in the pathogenesis of PCOS considering that changes in the content of DNA methylation, histone acetylation and noncoding RNAs have been reported in various tissues of women with this disease. DNA methylation is changed in the peripheral and umbilical cord blood, as well as in ovarian and adipose tissue of women with PCOS, suggesting the involvement of this epigenetic modification in the pathogenesis of the disease. Perhaps, these defects in DNA methylation promote the deregulation of genes involved in inflammation, hormone synthesis and signaling and glucose and lipid metabolism. Research on the role of DNA methylation in the pathogenesis of PCOS is just beginning, and several issues await investigation. This review aims to provide an overview of current research focused on DNA methylation and PCOS, as well as discuss the perspectives regarding this topic.

Filiaciones:
Ricardo Vazquez-Martinez, Edgar:
 Univ Nacl Autonoma Mexico, Fac Quim, Inst Nacl Perinatol, Unidad Invest Reprod Humana, Ciudad De Mexico, Mexico

Ines Gomez-Viais, Yadira:
 Univ Nacl Autonoma Mexico, Fac Quim, Inst Nacl Perinatol, Unidad Invest Reprod Humana, Ciudad De Mexico, Mexico

Garcia-Gomez, Elizabeth:
 Consejo Nacl Ciencia & Tecnol CONACyT, Inst Nacl Perinatol, Unidad Invest Reprod Humana, Ciudad De Mexico, Mexico

 Unidad de Investigación en Reproducción Humana, Consejo Nacional de Ciencia y Tecnología (CONACyT), Instituto Nacional de Perinatología, Ciudad de México, Mexico

Reyes-Mayoral, Christian:
 Inst Nacl Perinatol Isidro Espinosa de los Reyes, Subdirecc Reprod Humana, Ciudad De Mexico, Mexico

 Subdirección de Reproducción Humana, Instituto Nacional de Perinatología ‘Isidro Espinosa de los Reyes’, Ciudad de México, Mexico

Reyes-Munoz, Enrique:
 Inst Nacl Perinatol Isidro Espinosa de los Reyes, Coordinac Endocrinol Ginecol & Perinatal, Ciudad De Mexico, Mexico

 Coordinación de Endocrinología Ginecológica y Perinatal, Instituto Nacional de Perinatología ‘Isidro Espinosa de los Reyes’, Ciudad de México, Mexico

Camacho-Arroyo, Ignacio:
 Univ Nacl Autonoma Mexico, Fac Quim, Inst Nacl Perinatol, Unidad Invest Reprod Humana, Ciudad De Mexico, Mexico

 Unidad de Investigación en Reproducción Humana, Instituto Nacional de Perinatología-Facultad de Química, Universidad Nacional Autónoma de México, Ciudad de México, Mexico

Cerbon, Marco:
 Univ Nacl Autonoma Mexico, Fac Quim, Inst Nacl Perinatol, Unidad Invest Reprod Humana, Ciudad De Mexico, Mexico

 Unidad de Investigación en Reproducción Humana, Instituto Nacional de Perinatología-Facultad de Química, Universidad Nacional Autónoma de México, Ciudad de México, Mexico
ISSN: 14701626
Editorial
BIO SCIENTIFICA LTD, EURO HOUSE, 22 APEX COURT WOODLANDS, BRADLEY STOKE, BRISTOL BS32 4JT, ENGLAND, Reino Unido
Tipo de documento: Review
Volumen: 158 Número: 1
Páginas: 27-40
WOS Id: 000466887200003
ID de PubMed: 30959484