S1P(1) receptor phosphorylation, internalization, and interaction with Rab proteins: effects of sphingosine 1-phosphate, FTY720-P, phorbol esters, and paroxetine


Por: Carlos Martinez-Morales, Juan, Teresa Romero-Avila, M., Reyes-Cruz, Guadalupe, Adolfo Garcia-Sainz, J.

Publicada: 21 dic 2018
Resumen:
Sphingosine 1-phosphate (S1P) and FTY720-phosphate (FTYp) increased intracellular calcium in cells expressing S1P1 mCherry-tagged receptors; the synthetic agonist was considerably less potent. Activation of protein kinase C by phorbol myristate acetate (PMA) blocked these effects. The three agents induced receptor phosphorylation and internalization, with the action of FTYp being more intense. S1P1 receptor-Rab protein (GFP-tagged) interaction was studied using FRET. The three agents were able to induce S1P1 receptor-Rab5 interaction, although with different time courses. S1P1 receptor-Rab9 interaction was mainly increased by the phorbol ester, whereas S1P1 receptor-Rab7 interaction was only increased by FTYp and after a 30-min incubation. These actions were not observed using dominant negative (GDP-bound) Rab protein mutants. The data suggested that the three agents induce interaction with early endosomes, but that the natural agonist induced rapid receptor recycling, whereas activation of protein kinase C favored interaction with late endosome and slow recycling and FTYp triggered receptor interaction with vesicles associated with proteasomal/ lysosomal degradation. The ability of bisindolylmaleimide I and paroxetine to block some of these actions suggested the activation of protein kinase C was associated mainly with the action of PMA, whereas G protein-coupled receptor kinase (GRK) 2 (GRK2) was involved in the action of the three agents.

Filiaciones:
Carlos Martinez-Morales, Juan:
 Univ Nacl Autonoma Mexico, Dept Biol Celular & Desarrollo, Inst Fisiol Celular, Ciudad Univ,Ap Postal 70-248, Mexico City 04510, DF, Mexico

Teresa Romero-Avila, M.:
 Univ Nacl Autonoma Mexico, Dept Biol Celular & Desarrollo, Inst Fisiol Celular, Ciudad Univ,Ap Postal 70-248, Mexico City 04510, DF, Mexico

Reyes-Cruz, Guadalupe:
 Inst Politecn Nacl, Ctr Invest & Estudios Avanzados, Dept Biol Celular, CINVESTAV, Ave Inst Politecn Nacl 2508, Mexico City, DF, Mexico

Adolfo Garcia-Sainz, J.:
 Univ Nacl Autonoma Mexico, Dept Biol Celular & Desarrollo, Inst Fisiol Celular, Ciudad Univ,Ap Postal 70-248, Mexico City 04510, DF, Mexico
ISSN: 01448463





BIOSCIENCE REPORTS
Editorial
Kluwer Academic Publishers, THIRD FLOOR, EAGLE HOUSE, 16 PROCTER STREET, LONDON WC1V 6 NX, ENGLAND, Estados Unidos America
Tipo de documento: Article
Volumen: 38 Número: 6
Páginas:
WOS Id: 000454266100113
ID de PubMed: 30366961