Sclerocornea-Microphthalmia-Aphakia Complex: Description of Two Additional Cases Associated With Novel FOXE3 Mutations and Review of the Literature


Por: Quiroz-Casian N., Chacon-Camacho O.F., Barragan-Arevalo T., Nava-Valdez J., Lieberman E., Salgado-Medina A., Navas A., Graue-Hernandez E.O., Zenteno J.C.

Publicada: 1 sep 2018
Categoría: Ophthalmology

Resumen:
Purpose:To describe 2 sporadic Mexican patients having congenital bilateral, total sclerocornea, aphakia, and microphthalmia associated with novel mutations in the FOXE3 gene.Methods:Two affected individuals with congenital bilateral, total sclerocornea, aphakia, and microphthalmia underwent detailed examinations including slit-lamp examination, visual acuity, and intraocular pressure measurements. Ocular ultrasonography and ultrasound biomicroscopy were performed. Genomic DNA was isolated from blood leukocytes in each subject, and molecular analysis of the FOXE3 gene was performed. For cosegregation analysis, presumable pathogenic variants were tested by Sanger sequencing in parental DNA.Results:Molecular screening of FOXE3 was performed in 2 cases with congenital bilateral, total sclerocornea, aphakia, and microphthalmia. In patient 1, genetic analysis demonstrated a novel homozygous c.291C>G (p.Ile97Met) FOXE3 pathogenic variant. In patient 2, compound heterozygosity for the novel c.387C>G (p.Phe129Leu) transversion and for the previously reported c.244A>G (p.Met82Val) transition, was recognized.Conclusions:The sclerocornea-microphthalmia-aphakia complex is a severe malformative ocular phenotype resulting from mutations in the FOXE3 transcription factor. To date, patients from at least 14 families with this uncommon ocular disorder have been described. The identification of 2 novel pathogenic variants in our patients expands the mutational spectrum in FOXE3-related congenital eye disorders. In addition, we performed a review of the clinical and genotypic characteristics of all published patients carrying biallelic FOXE3 mutations.

Filiaciones:
Quiroz-Casian N.:
 Department of Cornea and Refractive Surgery, Institute of Ophthalmology "Conde de Valenciana, Mexico City, Mexico

Chacon-Camacho O.F.:
 Department of Genetics, Institute of Ophthalmology "Conde de Valenciana, Mexico City, Mexico

Barragan-Arevalo T.:
 Department of Genetics, Instituto Nacional de Pediatría, Mexico City, Mexico

Nava-Valdez J.:
 Department of Genetics, Institute of Ophthalmology "Conde de Valenciana, Mexico City, Mexico

Lieberman E.:
 Department of Genetics, Instituto Nacional de Pediatría, Mexico City, Mexico

Salgado-Medina A.:
 Department of Genetics, Institute of Ophthalmology "Conde de Valenciana, Mexico City, Mexico

Navas A.:
 Department of Cornea and Refractive Surgery, Institute of Ophthalmology "Conde de Valenciana, Mexico City, Mexico

Graue-Hernandez E.O.:
 Department of Cornea and Refractive Surgery, Institute of Ophthalmology "Conde de Valenciana, Mexico City, Mexico

Zenteno J.C.:
 Department of Genetics, Institute of Ophthalmology "Conde de Valenciana, Mexico City, Mexico

 Department of Biochemistry, Faculty of Medicine, UNAM, Mexico City, Mexico
ISSN: 02773740
Editorial
LIPPINCOTT WILLIAMS & WILKINS, 530 WALNUT ST, PHILADELPHIA, PA 19106-3621 USA, Estados Unidos America
Tipo de documento: Review
Volumen: 37 Número: 9
Páginas: 1178-1181
WOS Id: 000443952300020
ID de PubMed: 29878917