beta-Cyclodextrin and oleic acid increase levels of dopamine and potentiates oxidative damage in young and adult rat brain


Por: Calderon Guzman, David, Osnaya Brizuela, Norma, Ortiz Herrera, Maribel, Juarez Olguin, Hugo, Valenzuela Peraza, Armando, Barragan Mejia, Gerardo

Publicada: 25 jul 2018
Resumen:
Background: Cyclodextrins are active pharmaceutical ingredients to treat neurological diseases by reducing neurotoxicity. The aim of this study was to test if combined consumption of beta-cyclodextrin (BCD) and Oleic acid (OA) potentiates brain antioxidant protection. Methods: Four groups of young Wistar rats, grouped in 6 animals each, were treated as follows: Group (G) 1, saline solution 0.9% (control); G2, BCD (0.7 g/kg); G3, OA (15 ml/kg); G4, BCD + OA. The same design was assayed for groups of adult rats. Treatments were daily administered by oral means for five consecutive days. On the last day of administration, brains of the animals were extracted to measure dopamine, 5-HIAA, glutathione (GSH), ATPase, Lipoperoxidation and H2O2. Results: Oleic acid and beta-cyclodextrin upgraded the levels of dopamine, 5-HIAA and lipid peroxidation and downgraded the concentrations of GSH and H2O2 in cortex, hemispheres (striatum) and cerebellum/medulla oblongata regions. Conclusions: The results of the present study suggest that combined use of oleic acid and beta-cyclodextrin may increase oxidative damage in brain regions and promote alteration in dopamine and 5-HIAA amines and hence, constitutes health risks among age of subjects.

Filiaciones:
Calderon Guzman, David:
 INP, Lab Neuroquim, Mexico City, DF, Mexico

Osnaya Brizuela, Norma:
 INP, Lab Neuroquim, Mexico City, DF, Mexico

Ortiz Herrera, Maribel:
 INP, Lab Bacteriol Expt, Mexico City, DF, Mexico

Juarez Olguin, Hugo:
 Univ Nacl Autonoma Mexico, INP, Fac Med, Lab Farmacol, Ave Iman 1,3rd Piso Colonia Cuicuilco CP, Mexico City 04530, DF, Mexico

Valenzuela Peraza, Armando:
 INP, Lab Neuroquim, Mexico City, DF, Mexico

Barragan Mejia, Gerardo:
 INP, Lab Neuroquim, Mexico City, DF, Mexico
ISSN: 1476511X
Editorial
BIOMED CENTRAL LTD, 236 GRAYS INN RD, FLOOR 6, LONDON WC1X 8HL, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 17 Número: 1
Páginas:
WOS Id: 000439708500002
ID de PubMed: 30045742

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