Proteomic and ultrastructural analysis of the effect of a new nitazoxanide-N-methyl-1H-benzimidazole hybrid against Giardia intestinalis


Por: Matadamas-Martinez, Felix, Castillo, Rafael, Hernandez-Campos, Alicia, Mendez-Cuesta, Carlos, de Souza, Wanderley, Gadelha, Ana Paula, Nogueda-Torres, Benjamin, Manuel Hernandez, Jose, Yepez-Mulia, Lilian

Publicada: 1 abr 2016
Categoría: Veterinary (miscellaneous)

Resumen:
In an effort to develop alternative drugs for the treatment of giardiasis our research group has synthesized and evaluated a novel nitazoxanide and N-methyl-IH-benzimidazole hybrid molecule, named CMC-20. It showed an IC50 of 0.010 mu M on Giardia intestinalis, lower than the IC50 values of 0.015, 0.037 and 1.224 mu M for nitazoxanide, albendazole and metronidazole, respectively. In addition, we report studies carried out on its mechanism of action and effect at the ultrastructural level on G. intestinalis. The proteomic analysis of trophozoites treated with CMC-20 revealed significant changes in the expression level of proteins of the cytoskeleton, alpha and beta tubulin, alpha-1, beta giardin and axoneme-associated protein, among other molecules. Ultra structural studies demonstrated that CMC-20 induces morphological changes on the parasite that loses its characteristic pear shape. Uncommon large bulbous structure at the flagella end, and parasites showing flange membrane bending and a concave depression in the ventral region, resembling an encystation process, were also observed. In addition, some apoptotic and autophagic-like features, such as membrane blebbing, intense vacuolation, chromatin condensation and multilamellar bodies were detected. Phosphatidylserine externalization was determined as an apoptotic marker by flow cytometry and immunofluorescence microscopy; however, a typical ladder-like DNA fragmentation profile was not detected. Although it was found that CMC-20 triggers the encystation process, damage to the cyst wall indicates loss of viability. (C) 2016 Elsevier Ltd. All rights reserved.

Filiaciones:
Matadamas-Martinez, Felix:
 Inst Politecn Nacl, Escuela Nacl Ciencias Biol, Dept Parasitol, Mexico City 11340, DF, Mexico

Castillo, Rafael:
 Univ Nacl Autonoma Mexico, Fac Quim, Dept Farm, Mexico City 04510, DF, Mexico

Hernandez-Campos, Alicia:
 Univ Nacl Autonoma Mexico, Fac Quim, Dept Farm, Mexico City 04510, DF, Mexico

Mendez-Cuesta, Carlos:
 Univ Nacl Autonoma Mexico, Fac Quim, Dept Farm, Mexico City 04510, DF, Mexico

de Souza, Wanderley:
 Univ Fed Rio de Janeiro, Lab Ultraestrutura Celular Hertha Meyer, Inst Biofis Carlos Chagas Filho, BR-21949900 Rio de Janeiro, Brazil

 Inst Nacl Metrol Qualidade & Tecnol, Metrol Aplicada Ciencias Vida, Rio De Janeiro, Brazil

Gadelha, Ana Paula:
 Univ Fed Rio de Janeiro, Lab Ultraestrutura Celular Hertha Meyer, Inst Biofis Carlos Chagas Filho, BR-21949900 Rio de Janeiro, Brazil

 Inst Nacl Metrol Qualidade & Tecnol, Metrol Aplicada Ciencias Vida, Rio De Janeiro, Brazil

Nogueda-Torres, Benjamin:
 Inst Politecn Nacl, Escuela Nacl Ciencias Biol, Dept Parasitol, Mexico City 11340, DF, Mexico

Manuel Hernandez, Jose:
 Ctr Invest & Estudios Avanzados IPN, Dept Biol Celular, Mexico City 07360, DF, Mexico

Yepez-Mulia, Lilian:
 Pediat Inst Mexicano Seguro Social, Unidad Invest Med Enfermedades Infecciosas & Para, Mexico City 06720, DF, Mexico
ISSN: 00345288
Editorial
Elsevier, THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, OXON, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 105 Número:
Páginas: 171-179
WOS Id: 000374369400031
ID de PubMed: 27033928

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