Nucleo-cytoplasmic transport of estrogen receptor alpha in breast cancer cells


Por: Tecalco-Cruz, Angeles C., Perez-Alvarado, Issis A., Ramirez-Jarquin, Josue O., Rocha-Zavaleta, Leticia

Publicada: 1 jun 2017
Categoría: Cell biology

Resumen:
Approximately 70% cases of breast cancers exhibit high expression and activity levels of estrogen receptor alpha (ERa), a transcription regulator that induces the expression of genes associated with cellular proliferation and survival. These nuclear functions of the receptor are associated with the development of breast cancer. However, ERa localization is not static, but rather, dynamic with continuous shuttling between the nucleus and the cytoplasm. Interestingly, both the nuclear import and export of ERa are modulated by several stimuli that include estradiol, antiestrogens, and growth factors. As ERa nuclear accumulation is critical to the regulation of gene expression, nuclear export of this receptor modulates the intensity and duration of its transcriptional activity. Thus, the subcellular spatial distribution of ERa ensures tight modulation of its concentration in cellular compartMents, as well as of its nuclear and extranuclear functions. In this review, we will discuss current findings regarding the biological importance of molecular mechanisms of, and proteins responsible for, the nuclear import and export of ERa in breast cancer cells.

Filiaciones:
Tecalco-Cruz, Angeles C.:
 Univ Nacl Autonoma Mexico, Inst Invest Biomed, Programa Invest Canc Mama, Apdo Postal, Mexico City 04510, DF, Mexico

Perez-Alvarado, Issis A.:
 Univ Nacl Autonoma Mexico, Inst Invest Biomed, Programa Invest Canc Mama, Apdo Postal, Mexico City 04510, DF, Mexico

Ramirez-Jarquin, Josue O.:
 Univ Nacl Autonoma Mexico, Inst Fisiol Celular, Apdo Postal, Mexico City 04510, DF, Mexico

Rocha-Zavaleta, Leticia:
 Univ Nacl Autonoma Mexico, Inst Invest Biomed, Programa Invest Canc Mama, Apdo Postal, Mexico City 04510, DF, Mexico
ISSN: 08986568
Editorial
Elsevier Inc., 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA, Estados Unidos America
Tipo de documento: Review
Volumen: 34 Número:
Páginas: 121-132
WOS Id: 000401388600013
ID de PubMed: 28341599