Dual regulation of the T-type Ca2+ current by serum albumin and ß- estradiol in mammalian spermatogenic cells


Por: Espinosa F., López-González I., Muñoz-Garay C., Felix R., De La Vega-Beltrán J.L., Kopf G.S., Visconti P.E., Darszon A.

Publicada: 1 ene 2000
Resumen:
This study provides evidence for a novel mechanism of voltage-gated Ca2+ channel regulation in mammalian spermatogenic cells by two agents that affect sperm capacitation and the acrosome reaction (AR). Patch-clamp experiments demonstrated that serum albumin induced an increase in Ca2+ T current density in a concentration-dependent manner, and significant shifts in the voltage dependence of both steady-state activation and inactivation of the channels. These actions were not related to the ability of albumin to remove cholesterol from the membrane. In contrast, ?-estradiol significantly inhibited Ca2+ channel activity in a concentration-dependent and essentially voltage-independent fashion. In mature sperm this dual regulation may influence capacitation and/or the AR. (C) 2000 Federation of European Biochemical Societies.

Filiaciones:
Espinosa F.:
 Dept. of Genet. and Molec. Physiol., Inst. Biotech., UNAM, Ave. U., Cuernavaca, Mexico

López-González I.:
 Dept. of Genet. and Molec. Physiol., Inst. Biotech., UNAM, Ave. U., Cuernavaca, Mexico

Muñoz-Garay C.:
 Dept. of Genet. and Molec. Physiol., Inst. Biotech., UNAM, Ave. U., Cuernavaca, Mexico

Felix R.:
 Dept. of Genet. and Molec. Physiol., Inst. Biotech., UNAM, Ave. U., Cuernavaca, Mexico

 Department of Physiology, Biophys. Neurosci., Cinvestav-IPN, Mexico City, Mexico

De La Vega-Beltrán J.L.:
 Dept. of Genet. and Molec. Physiol., Inst. Biotech., UNAM, Ave. U., Cuernavaca, Mexico

Kopf G.S.:
 Ctr. Res. Repro. and Women's Hlth., Univ. of Pennsylvania Medical Center, Philadelphia, PA, United States

Visconti P.E.:
 Dept. of Anatomy and Cell Biology, University of Virginia, Charlottesville, VA, United States

Darszon A.:
 Dept. of Genet. and Molec. Physiol., Inst. Biotech., UNAM, Ave. U., Cuernavaca, Mexico
ISSN: 00145793
Editorial
Elsevier, PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS, Estados Unidos America
Tipo de documento: Article
Volumen: 475 Número: 3
Páginas: 251-256
WOS Id: 000087931700020
ID de PubMed: 10869566
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