Humoral immunity in the gut selectively targets phenotypically virulent attaching-and-effacing bacteria for intraluminal elimination


Por: Kamada N., Sakamoto K., Seo S.-U., Zeng M.Y., Kim, YG, Cascalho M., Vallance B.A., Puente J.L., Nunez, G

Publicada: 13 may 2015
Resumen:
Virulence factors expressed by enteric bacteria are pivotal for pathogen colonization and induction of intestinal disease, but the mechanisms by which host immunity regulates pathogen virulence are largely unknown. Here we show that specific antibody responses are required for downregulation of virulence gene expression in Citrobacter rodentium, an enteric pathogen that models human infections with attaching-and-effacing bacteria. In the absence of antibodies against the pathogen, phenotypically virulent C. rodentium, accumulated and infected the epithelium and subsequently invaded the lamina propia, causing host lethality. IgG induced after infection recognized virulence factors and bound virulent bacteria within the intestinal lumen, leading to their engulfment by neutrophils, while phenotypically avirulent pathogens remained in the intestinal lumen and were eventually outcompeted by the microbiota. Thus, the interplay of the innate and adaptive immune system selectively targets virulent C. rodentium in the intestinal lumen to promote pathogen eradication and host survival. © 2015 Elsevier Inc.

Filiaciones:
Kamada N.:
 Division of Gastroenterology, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, MI 48109, United States

Sakamoto K.:
 Department of Pathology and Comprehensive Cancer Center, University of Michigan Medical School, Ann Arbor, MI 48109, United States

Seo S.-U.:
 Department of Pathology and Comprehensive Cancer Center, University of Michigan Medical School, Ann Arbor, MI 48109, United States

Zeng M.Y.:
 Department of Pathology and Comprehensive Cancer Center, University of Michigan Medical School, Ann Arbor, MI 48109, United States

Cascalho M.:
 Department of Surgery and Microbiology and Immunology, University of Michigan Medical School, Ann Arbor, MI 48109, United States

Vallance B.A.:
 Division of Gastroenterology, BC Children's Hospital, University of British Columbia, Vancouver, BC, Canada

Puente J.L.:
 Univ Nacl Autonoma Mexico, Inst Biotecnol, Dept Microbiol Mol, Cuernavaca 62210, Morelos, Mexico
ISSN: 19313128
Editorial
Cell Press, 600 TECHNOLOGY SQUARE, 5TH FLOOR, CAMBRIDGE, MA 02139 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 17 Número: 5
Páginas: 617-627
WOS Id: 000356101500013
ID de PubMed: 25936799
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