Certain Strongylocentrotus purpuratus sperm mitochondrial proteins co-purify with low density detergent-insoluble membranes and are PKA or PKC-substrates possibly involved in sperm motility regulation


Por: Loza-Huerta, A, Vera-Estrella, R, Darszon, A, Beltran, C

Publicada: 1 nov 2013
Resumen:
Background: Sea urchin sperm motility is regulated by Speract, a sperm-activating peptide (SAP) secreted from the outer egg coat. Upon binding to its receptor in the sperm flagellum, Speract induces a series of ionic and metabolic changes in Strongylocentrotus purpuratus spermatozoa that regulate their motility. Among these events, protein phosphorylation is one of the most relevant and evidence indicates that some proteins of the Speract signaling cascade localize in low density detergent-insoluble membranes (LD-DIM). Methods: LD-DIM-derived proteins from immotile, motile or Speract-stimulated S. purpuratus sperm were resolved in 2-D gels and the PKA and PKC substrates detected with specific antibodies were identified by LCMS/MS. Results: Differential PICA and PKC substrate phosphorylation levels among the LD-DIM isolated from sperm in different motility conditions were foiind and identified by mass spectrometry as: ATP synthase, creatine kinase, NADH dehydrogenase (ubiquinone) flavoprotein 2, succinyl-CoA ligase and the voltage-dependent anion channel 2 (VDAC2), which are mitochondrial proteins, as well as, the cAMP-dependent protein kinase type II regulatory (PICA RII) subunit, Tubulin beta chain and Actin Cy I changed their phosphorylation state. Conclusions: Some mitochondrial proteins regulated by PICA or PKC may influence sea urchin sperm motility. General significance: The fact that a high percentage (66%) of the PICA or PKC substrates identified in LD-DIM are mitochondrial proteins suggests that the phosphorylation of these proteins modulates sea urchin sperm motility via Speract stimulation by providing sufficient energy to sperm physiology. Those mitochondrial proteins are indeed PICA- or PKC-substrates in the sea urchin spermatozoa. (C) 2013 Elsevier B.V. All rights reserved.

Filiaciones:
Loza-Huerta, A:
 Univ Nacl Autonoma Mexico, Dept Genet Desarrollo & Fisiol Mol, Cuernavaca 62210, Morelos, Mexico

Vera-Estrella, R:
 Univ Nacl Autonoma Mexico, Inst Biotecnol, Cuernavaca 62210, Morelos, Mexico

Darszon, A:
 Univ Nacl Autonoma Mexico, Dept Genet Desarrollo & Fisiol Mol, Cuernavaca 62210, Morelos, Mexico

Beltran, C:
 Univ Nacl Autonoma Mexico, Dept Genet Desarrollo & Fisiol Mol, Cuernavaca 62210, Morelos, Mexico
ISSN: 03044165
Editorial
ELSEVIER SCIENCE BV, PO BOX 211, 1000 AE AMSTERDAM, NETHERLANDS, Países Bajos
Tipo de documento: Article
Volumen: 1830 Número: 11
Páginas: 5305-5315
WOS Id: 000326207600036
ID de PubMed: 23928041