A noncanonical NF-kappa B pathway through the p50 subunit regulates Bcl-2 overexpression during an oxidative-conditioning hormesis response


Por: Luna-López A., González-Puertos V.Y., Romero-Ontiveros J., Ventura-Gallegos J.L., Zentella A., Gomez-Quiroz L.E., Konigsberg, M

Publicada: 1 oct 2013
Resumen:
Cells can respond to damage and stress by activating various repair and survival pathways. One of these responses can be induced by preconditioning the cells with sublethal stress to provoke a prosurvival response that will prevent damage and death, and which is known as hormesis. Bcl-2, an antiapoptotic protein recognized by its antioxidant and prosurvival functions, has been documented to play an important role during oxidative-conditioning hormesis. Using an oxidative-hormetic model, which was previously established in the L929 cell line by subjecting the cells to a mild oxidative stress of 50 mu M H2O2 for 9 h, we identified two different transductional mechanisms that participate in the regulation of Bcl-2 expression during the hormetic response. These mechanisms converge in activating the nuclear transcription factor NF-kappa B. Interestingly, the noncanonical p50 subunit of the NF-kappa B family is apparently the subunit that participates during the oxidative-hormetic response. (C) 2013 Elsevier Inc. All rights reserved.

Filiaciones:
Luna-López A.:
 Instituto Nacional de Geriatría, SSA, México, DF 10200, Mexico

González-Puertos V.Y.:
 Departamento de Ciencias de la Salud, División de Ciencias Biológicas y de la Salud, Universidad Autónoma Metropolitana Iztapalapa, México, DF 09340, Mexico

Romero-Ontiveros J.:
 Departamento de Ciencias de la Salud, División de Ciencias Biológicas y de la Salud, Universidad Autónoma Metropolitana Iztapalapa, México, DF 09340, Mexico

Ventura-Gallegos J.L.:
 Univ Nacl Autonoma Mexico, IIB, Dept Med Genom & Toxicol Ambiental, Mexico City 04510, DF, Mexico

Zentella A.:
 Univ Nacl Autonoma Mexico, IIB, Dept Med Genom & Toxicol Ambiental, Mexico City 04510, DF, Mexico

Gomez-Quiroz L.E.:
 Departamento de Ciencias de la Salud, División de Ciencias Biológicas y de la Salud, Universidad Autónoma Metropolitana Iztapalapa, México, DF 09340, Mexico
ISSN: 08915849
Editorial
ELSEVIER SCIENCE INC, 360 PARK AVE SOUTH, NEW YORK, NY 10010-1710 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 63 Número:
Páginas: 41-50
WOS Id: 000323094700003
ID de PubMed: 23648765

MÉTRICAS