Endogenous Content and Release of [H-3]-GABA and [H-3]-Glutamate in the Spinal Cord of Chronically Undernourished Rat


Por: Quiroz-Gonzalez, S, Escartin-Perez, RE, Paz-Bermudez, F, Segura-Alegria, B, Reyes-Legorreta, C, Guadarrama-Olmos, JC, Floran-Garduno, B, Jimenez-Estrada, I

Publicada: 1 ene 2013
Resumen:
The aim of this study was to determine the effect of chronic undernutrition on the content and release of gamma-amino butyric acid (GABA) and glutamate (GLU) transmitters in the rat spinal cord. The release of [H-3]-GABA and [H-3]-GLU was determined by radioactive liquid scintillation techniques, and the concentrations of GABA and GLU in spinal cord preparations from control and undernourished young rats (50-60 days old) were measured by reverse-phase HPLC. The GABA and GLU contents in the lumbar spinal dorsal horn (L6 segment) were significantly lower in undernourished rats relative to control rats (22.2 +/- A 3.7 and 10.7 +/- A 1.9 %, respectively; P < 0.05). Spinal cord blocks from undernourished animals also showed lower rates of [H-3]-GABA and [H-3]-GLU release than controls (27.6 +/- A 3.5 and 12.8 +/- A 2.5 %, respectively; P < 0.01). We propose that the decreases in GLU content and release are consistent with a reduced activation of either afferent fibers, spinal glutaminergic neurons, or both. Furthermore, we propose that the decreased content and release of GABA in undernourished animals are related to a depression in pre- and post-synaptic inhibition. In addition, we hypothesize that the reductions in GABA content and release serve as compensatory mechanisms to counterbalance decreases in sensory transmission and GLU content in the spinal cord of the chronically undernourished rat.

Filiaciones:
Escartin-Perez, RE:
 Univ Nacl Autonoma Mexico, Lab Neurobiol Alimentac, FES Iztacala, Mexico City, DF, Mexico

Segura-Alegria, B:
 Univ Nacl Autonoma Mexico, Fac Estudios Super, FES Iztacala, Mexico City, DF, Mexico
ISSN: 03643190
Editorial
SPRINGER/PLENUM PUBLISHERS, 233 SPRING ST, NEW YORK, NY 10013 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 38 Número: 1
Páginas: 23-31
WOS Id: 000312664400002
ID de PubMed: 22983619