PATHWAYS INVOLVED IN THE GENERATION OF REACTIVE OXYGEN AND NITROGEN SPECIES DURING GLUCOSE DEPRIVATION AND ITS ROLE ON THE DEATH OF CULTURED HIPPOCAMPAL NEURONS


Por: Paramo, B, Hernandez-Fonseca, K, Estrada-Sanchez, AM, Jimenez, N, Hernandez-Cruz, A, Massieu, L

Publicada: 2 jun 2010
Categoría: Neuroscience (miscellaneous)

Resumen:
Oxidative stress has been suggested as a mechanism contributing to neuronal death induced by hypoglycemia, and an early production of reactive species (RS) during the hypoglycemic episode has been observed. However, the sources of reactive oxygen (ROS) and nitrogen (RNS) species have not been fully identified. In the present study we have examined the contribution of various enzymatic pathways to RS production and neuronal death induced by glucose deprivation (GD) in hippocampal cultures. We have observed a rapid increase in RS during GD, which depends on the activation of NMDA and non-NMDA receptors and on the influx of calcium from the extracellular space. Accordingly, intracellular calcium concentration [Ca(2+)](i) progressively increases more than 30-fold during the GD period. It was observed that superoxide production through the activation of the calcium-dependent enzymes, phospholipase A(2) (cPLA(2)) and xanthine oxidase (XaO), contributes to neuronal damage, while nitric oxide

Filiaciones:
Paramo, B:
 Univ Nacl Autonoma Mexico, Inst Fisiol Celular, Dept Neuropatol Mol, Div Neurociencias, Mexico City 04510, DF, Mexico

Hernandez-Fonseca, K:
 Univ Nacl Autonoma Mexico, Inst Fisiol Celular, Dept Neuropatol Mol, Div Neurociencias, Mexico City 04510, DF, Mexico

Estrada-Sanchez, AM:
 Univ Nacl Autonoma Mexico, Inst Fisiol Celular, Dept Neuropatol Mol, Div Neurociencias, Mexico City 04510, DF, Mexico

Jimenez, N:
 Univ Nacl Autonoma Mexico, Inst Fisiol Celular, Dept Neurociencia Cognit, Mexico City 04510, DF, Mexico

Hernandez-Cruz, A:
 Univ Nacl Autonoma Mexico, Inst Fisiol Celular, Dept Neurociencia Cognit, Mexico City 04510, DF, Mexico

Massieu, L:
 Univ Nacl Autonoma Mexico, Inst Fisiol Celular, Dept Neuropatol Mol, Div Neurociencias, Mexico City 04510, DF, Mexico
ISSN: 03064522





Neuroscience
Editorial
PERGAMON-ELSEVIER SCIENCE LTD, THE BOULEVARD, LANGFORD LANE, KIDLINGTON, OXFORD OX5 1GB, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 167 Número: 4
Páginas: 1057-1069
WOS Id: 000277434800011
ID de PubMed: 20226235