Rituximab-Mediated Cell Signaling and Chemo/Immuno-sensitization of Drug-Resistant B-NHL Is Independent of Its Fc Functions


Por: Vega M.I., Huerta-Yepez S., Martinez-Paniagua M., Martinez-Miguel B., Hernandez-Pando R., González-Bonilla C.R., Chinn P., Hanna N., Hariharan K., Jazirehi A.R., Bonavida B.

Publicada: 1 nov 2009
Resumen:
Purpose: Rituximab [chimeric anti-CD20 monoclonal antibody], alone or combined with chemotherapy, is used in the treatment of non-Hodgkin's lymphoma (NHL). Rituximab binds to CD20 and inhibits intracellular survival/growth pathways leading to chemo/immunosensitization of tumor cells in vitro. The contribution of rituximab Fc-FcR interaction in signaling is not known. This study examined the role of Fc-FcR interactions in rituximab-induced signaling using rituximab (Fab')2 fragments as well as rituximab devoid of the CH2 Fc-binding domain (CH2(-)). Experimental Design: Rituximab (CH2(-)) and rituximab (Fab')(2) were tested for their activity on B-NHL cell lines. Cell signaling and sensitization to chemotherapy and immunotherapy were examined. The in vitro studies were validated in mice bearing tumor xenografts. Results: Although the modified antibodies were defective in antibody-dependent cellular cytotoxicity and complement-dependent cytotoxicity functions, they retained all other biol

Filiaciones:
Vega M.I.:
 Department of Microbiology, Immunology, and Molecular Genetics, David Geffen School of Medicine, University of California, Los Angeles, CA, United States

 Unidad de Investigación Médica en Inmunología e Infectología, Hospital de Infectología, CMN La Raza, México City, Mexico

Huerta-Yepez S.:
 Department of Microbiology, Immunology, and Molecular Genetics, David Geffen School of Medicine, University of California, Los Angeles, CA, United States

 Unidad de Investigación Médica en Inmunología e Infectología, Hospital de Infectología, CMN La Raza, México City, Mexico

Martinez-Paniagua M.:
 Unidad de Investigación Médica en Inmunología e Infectología, Hospital de Infectología, CMN La Raza, México City, Mexico

Martinez-Miguel B.:
 Unidad de Investigación Médica en Inmunología e Infectología, Hospital de Infectología, CMN La Raza, México City, Mexico

Hernandez-Pando R.:
 Sección de Patología Experimental, Departamento de Patología, Instituto Nacional de Ciencias Médicas y Nutrición Salvador Zubirán, Mexico City, Mexico

González-Bonilla C.R.:
 Unidad de Investigación Médica en Inmunología e Infectología, Hospital de Infectología, CMN La Raza, México City, Mexico

Chinn P.:
 Biogen-Idec, Inc., San Diego, CA, United States

Hanna N.:
 Biogen-Idec, Inc., San Diego, CA, United States

Hariharan K.:
 Biogen-Idec, Inc., San Diego, CA, United States

Jazirehi A.R.:
 Department of Microbiology, Immunology, and Molecular Genetics, David Geffen School of Medicine, University of California, Los Angeles, CA, United States

Bonavida B.:
 Department of Microbiology, Immunology, and Molecular Genetics, David Geffen School of Medicine, University of California, Los Angeles, CA, United States
ISSN: 10780432
Editorial
AMER ASSOC CANCER RESEARCH, 615 CHESTNUT ST, 17TH FLOOR, PHILADELPHIA, PA 19106-4404 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 15 Número: 21
Páginas: 6582-6594
WOS Id: 000271300200015
ID de PubMed: 19861448
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